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## Chapter 13 ## Getting Started with Photoshop
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St. Clement of Rome Catholic School, Omaha St. Clement of Rome Catholic School is a private, coeducational, non-profit Catholic school located in Omaha, Nebraska. It is located within the Roman Catholic Diocese of Omaha. History St. Clement of Rome was founded in 1878 by the Rev. Herman Albers and became a parochial school in 1881. In 1905, the church of St. Clement was dedicated and the current main building was constructed. The Rev. George M. Heicklen was appointed pastor in 1909 and established the school with 12 students and 9 teachers. Academics Academics at St. Clement include the St. Clement Diploma program, college prep and Catholic high school Diploma program, and elementary diploma program. See also St. Clement of Rome Church (Omaha) References Category:Roman Catholic Diocese of Omaha Category:Catholic secondary schools in Nebraska Category:Schools in Omaha, Nebraska Category:Educational institutions established in 1878 Category:1878 establishments in NebraskaQ: Python – Pseudocode for a Matrix “Get” Function So I was wondering, If there was a basic function, similar to arrays, where you could call a function like, a = A(b=1, x=2) and it would “get” information from the value you gave to the function. a[‘x’] #returns 2 a[‘x’] = 3 #value changes to 3 So basically, I’m looking for how to make a basic matrix class that would be able to get a value. It could be done using the variables in the class (unlikely), or by making a function, and passing it a number of the variables So, class Matrix: def get(val): #call function passing in val a = Matrix(x=1, y=2) a.get(y) #returns 2 a.get(x) #returns 2 a.get(x=3) #returns 3 Is there anyway that this could be done in real life? EDIT: More info The main goal is to be able to use it with numpy matrices like a = np.matrix([[1,2],[3,4]])
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Alopecia areata: current understanding and potential therapeutic uses of systemic cytokines. Alopecia areata (AA) is a commonly occurring hair loss disease of unknown etiology. It affects millions of people worldwide and is highly prevalent among children and women. Epidemiological studies have identified herpes simplex virus type 1 and the mammalian epidermal growth factor receptor as etiological factors, while there is evidence for genetic susceptibility factors. However, the pathogenesis of the disease is still unknown. AA is generally considered to be an autoimmune disease of the skin in which genetic susceptibility and environmental factors are important determinants of disease expression. Cytokines, proteins produced by the immune system, can modulate the immune response to foreign antigens, and abnormalities in cytokine production have been linked with many immune disorders. The aim of the present study was to evaluate the occurrence of AA in patients with various immune disorders (rheumatoid arthritis, psoriasis, systemic lupus erythematosus, and myasthenia gravis), to compare the serum cytokine levels in AA and normal control groups, and to examine whether the serum levels of cytokines would be affected by immunotherapy. Twelve patients with moderate AA were enrolled. Serum levels of tumour necrosis factor alpha (TNF-alpha), interferon gamma (IFN-gamma), interleukin 10 (IL-10), and transforming growth factor beta (TGF-beta) were determined using enzyme-linked immunosorbent assay (ELISA). Serum levels of TNF-alpha and TGF-beta were higher in the AA group compared with the control group (P < 0.05). The levels of IL-10 and IFN-gamma did not differ significantly between the two groups. There were no correlations between TNF-alpha, TGF-beta, IFN-gamma, IL-10, TNF-alpha, and TGF-beta levels and clinical parameters of AA. IL-10 and IFN-gamma were lower in the clinical response group than in the non-response group (P < 0.05). Serum TNF-alpha, TGF-beta, IFN-gamma, and IL-10 levels in AA were increased compared with controls and did not correlate with clinical and laboratory findings. However, TGF-beta levels were significantly higher in the responders group than in the non-responders group. Since TGF-beta is known
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